Compound Profile

TB 500

Synthetic thymosin beta-4 fragment

TB 500 is a synthetic fragment of thymosin beta 4 marketed for muscle recovery and connective tissue repair; the underlying Tβ4 biology has real scientific grounding, but the synthetic fragment sold as TB 500 has thin direct human evidence and an unresolved FDA compounding status pending July 2026 PCAC review.

Use extra caution No controlled human safety data exists for the synthetic TB 500 fragment. Details

Last reviewed: Report a correctionProfile-wide ·D Preliminary
TB 500 monograph visual
Generated profile category visual

At a glance

From this profile's structured data
Evidence
Grade D
Safety
No controlled human safety data exists for the synthetic TB-500 fragment.
Profile context
Actin (G-actin) Binding, Cell Migration Promotion, Angiogenesis Support
Next steps

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Unapproved compound: FDA safety and compounding review

TB-500 is not FDA-approved for human use. FDA’s Pharmacy Compounding Advisory Committee discussed the thymosin-beta-4 fragment on July 23, 2026; that advisory process does not itself create an approved drug or a final 503A bulks-list status.

  • FDA says it has not identified human exposure data for the TB-500 fragment in its current compounding safety review.
  • This profile is not a self-treatment guide.
  • A research-use label does not establish FDA-reviewed identity, quality, safety, or effectiveness for human use.

Regulatory status

2026 federal and state regulatory context

Last checked 2026 06 30

Not FDA approved for human use; RUO labeled product is not legally intended for human consumption.

Regulatory changelog

  • 2023 04/2026: FDA Category 2 restricted (503A bulk substances), alongside BPC 157 and 17 other peptides. 2026 04: Removed from Category 2 (gray zone). 2026 07 23: On FDA PCAC agenda alongside BPC 157, KPV, and MOTS C for formal 503A eligibility review.

Quick stats

Evidence level
Preliminary evidence
Typical onset
Varies by prep
Safety rating
Use extra caution: Use caution
Best for
Actin (G Actin) Binding, Cell Migration Promotion, Angiogenesis Support
Avoid / review if
Pregnancy/Breastfeeding, Unsupervised RUO Product Use Without Physician Oversight, No Established Pharmacokinetic Drug Interaction Data Exists

Safety

Safety & Cautions

No controlled human safety data exists for the synthetic TB 500 fragment. Because Tβ4 supports angiogenesis and cell migration broadly, some researchers urge caution in anyone with a personal or family history of cancer, though this has not been demonstrated for TB 500 in humans.

High caution

Caution level: High. Multiple caution categories apply, or the record uses avoid/contraindication language.

  • Interaction-AwareMedication or interaction context is explicitly noted.
  • Pregnancy/Breastfeeding CautionPregnancy or breastfeeding caution language is present.

Evidence Summary

Profile-wide ·D Preliminary

Evidence lens

Early signal that needs stronger human replication before practical claims.

PreliminaryPreliminary evidence

Human clinical evidence: Not the primary signal

Mechanistic / preclinical: Mechanism mappedActin (G Actin) Binding · Cell Migration Promotion · Angiogenesis Support

Research maturity: Theoretical / mechanistic researchMechanistic or unresolved evidence is kept separate from established human outcomes.

Safety boundary: Safety note availableNo controlled human safety data exists for the synthetic TB 500 fragment.

Evidence Strength

Confidence estimate based on the design quality and consistency of published clinical trials.

Preliminary evidence

TB 500 has a preliminary evidence rating.

PerformanceRecovery
Effects
4
How evidence grades work

Each grade reflects the strength and consistency of published human evidence — not marketing claims. Grades are based on study count, design quality, effect size, consistency, and recency.

Strong
Multiple RCTs, consistent direction, adequate effect size
Moderate
Some RCTs or consistent observational data in humans
Preliminary / Mixed
Animal or in-vitro only, or conflicting human data
Traditional / Theoretical
Traditional use only; no controlled human trials

How TB 500 Works

Simplified mechanism pathway based on preclinical and pharmacological evidence. Does not confirm clinical efficacy.

Mechanism Pathway — How TB 500 Works

How TB 500 WorksTB 500 acts on biological target pathways via angiogenesis (modulation), leading to actin (g actin) binding.TB 500Biological TargetBiological pathwayAngiogenesisModulationActin (G Actin) BindingObservable outcome
CompoundTarget / ReceptorMechanismEffect / Outcome

Dosing

No standardized dose
No established consumer dosing protocol; see full guide article for regulatory and safety context.
Mechanisms & biological pathways

Preclinical mechanism details from scientific profiles; these represent plausible pathways but do not guarantee clinical efficacy in humans.

  • Actin (G Actin) Binding
  • Cell Migration Promotion
  • Angiogenesis Support
  • Anti Fibrotic Signaling
  • Cell Migration
  • Angiogenesis

Compare & Sourcing

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Sourcing options disabled for safety

Direct product recommendations and affiliate links are suppressed for this compound due to its high caution or needs-review safety classification.

Evaluate the safety checks, contraindications, and potential medication interactions above under clinician supervision before use.

Safety first · Harm reduction

This information is educational and not medical advice. Start low, avoid risky combinations, and consult a licensed clinician before making health decisions—especially if you use medications, have diagnosed conditions, or are pregnant/breastfeeding.

Learn how we evaluate confidence, safety, and intensity on the methodology page.

Editorial Review

Checked against primary sources, cited evidence, and contraindication language before publication. Evidence claims, safety language, and affiliate modules are reviewed independently. Not personal medical advice.

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Compound profile context

How to interpret TB 500

TB 500 compound profile with evidence context, key research topics, source notes, safety context, and references presented in an evidence-first format. Use this compound profile to understand mechanism, evidence quality, and practical safety context before comparing products or building a stack. A biochemical pathway connection can be useful for discovery, but it is not the same as proven benefit in humans.

When reviewing TB 500, separate the active molecule from the product category around it. Dose form, absorption, extract standardization, medication interactions, and individual sensitivity can change the real-world risk-benefit picture.

For supplement planning, avoid combining multiple compounds with overlapping sedating, stimulating, serotonergic, blood-pressure, anticoagulant, or liver-metabolism effects unless a qualified clinician has reviewed the context.